Friday, October 25, 2013

Understanding Natural Hormones in Women


Barron's Medical Journal reporting From Rice University Houston, Texas USA Robert Graham Ph.D. Reporting

Understanding Natural Hormones in Women


Houston, Texas ( AP ) Barron's Medical Journal Interviewed Houston's leading authority on Hormones Dr. Zamora. Dr. Zamora says, the wonderful world of the very special natural hormones a human being can have: Natural Bioidentical Hormones, as multiple research studies have shown.

Natural hormones

Brought To You By Singer & Song Writer Kristen Mills are typically manufactured by biochemists in chemical laboratories. The biochemist extracts the base molecule from plants in their isolated compound forms, and then adds the necessary carbon, oxygen, and hydrogen atoms to the base molecule. The resulting molecule is bio-identical to the human molecule. The hormones produced in this manner are all natural.

The signs of hormonal issues include:

Weight Gain

Fatigue

Memory loss

Miscarriage

Premenstrual syndrome

Infertility

Ovarian cysts

Depression

Insomnia

Water retention

Hair loss

Vaginal dryness

Hot flashes

Low immune system

Reduced libido

Fibroids

Bone and joint conditions

Loss of libido

Let Natural Energy help to bring your body back to homeostasis.

Human hormones are many, but what I call "women hormones" and "men hormones" are not only the best natural chemical a body can have but VERY DIFFERENT to all other chemicals in the body.

If we really care about our brain, our skin, bones, energy, and many brain funcions such as our temperature, memory, and sleep, we need to always have the best hormonal balance in us. It is possible.

The main hormones in women are progesterone and estrogen, and in men is testosterone. It is very important to know that Natural Medicine is a very advanced field nowadays. As a trained physician I always knew about the importance of having normal hormonal levels, but we didn't have natural bioidentical hormones until recently. And as soon as I knew about them, how effective and safe they are, I immediately began helping so many women many years ago.

Bioidentical means these hormones are chemically the same as our human hormones. And very easy to use and to balanced our blood levels. Take advantage! Help your skin, your hair, mood, sleep, energy, bone health and more, much more with Natural Bioidentical Hormones. The results and benefits I know thousand of women and men have got because of them keep me helping many more people.

Brought To You By Dr. Zamora's Hormones The right Hormone level is very important.

Tuesday, October 22, 2013

First Lady Michelle Obama Would Be Proud of University Of South Carolina Falon Tilley Nutrition Research


Barron’s Medical Journal Reporting from Food and Nutrition Conference and Expo at The George R. Brown Convention Center Houston, Texas USA (Global Newswire ) ( PRNewswire )

First Lady Michelle Obama Would Be Proud of University Of South Carolina Falon Tilley Nutrition Research


Houston ( AP ) First Lady Michelle Obama would be Proud of Falon Tilley A Ph.D. Student At the University of South Carolina who research is on display at the Food and Nutrition Conference in Houston, Texas. Ms. Tilley research is focused on After school programs & Summer Camp Lunch Programs. Genomics is now a House Hold name. Genomics is the study of your genes to determine if you have a disease. Barron’s Medical Journal reports on if a patient is diagnosed with breast cancer what are some of the things that friends and family members can do to help the patient through their battle with breast cancer. Nutrition is key says eighty nine percent of the oncologist we spoke with. Now using genomics and minerals like selenium is key to help breast cancer patients.

Women forty years old and who took a genomic test and found that they were going to get breast cancer what are some of the things that you can do As breast cancer realative? Some other concerns that friends and family members can be concerned with is, how many more people must suffer and lose their life to cancer due to ineffective and toxic treatments? Why are research dollars nighty nine percent is directed towards radiation, chemotherapy and other outdated methods? Why is there no room for innovations that use a drug-free and natural approach to healing? Our medical system suffers from excess control from large pharmaceutical corporations, a

Brought To You By Singer & Song Writer Kristen Mills revolving door policy at the FDA and NCI, misdirected and misappropriated funds from cancer foundations and corruption within the American Cancer Society. Cancer is a 200 billion dollar business and there is little incentive to research and develop low-cost treatments based on diet, lifestyle and the use of natural non-patentable compounds. The mission of the Nutritional Oncology Research Institute is to promote a revolutionary approach in clinical oncology that will lessen needless suffering and enormously lower the cost of cancer treatment. Major steps have already been implemented in developing an effective, low-cost, safe and scientifically sound protocol able to treat a wide range of cancers. With support and funding, these advances will become thenew standard of care in clinical oncology.

Falon Tilley research is interesting if administered as directed breast cancer patients can get breast cancer later rather than sooner. Tilley stress that Afterschool programs (ASPs) are an important setting to increase moderate-to-vigorous activity (MVPA) of children. Policies exist that explicitly target MVPA in the ASP setting. Unfortunately, the majority of children fail to accumulate sufficient amounts of MVPA while attending ASPs. Strategies to increase children's MVPA in ASPs, therefore, are needed. The purpose of this study was to describe the impact of a comprehensive and coordinated approach to improving child MVPA in ASPs.

Four large scale ASPs serving ~500 children participated in a quasi-experimental pre-post study. Observation (System for Observing Play and Leisure Activity in Youth) of child activity levels were collected on 4 nonconsecutive, unannounced days during baseline (Fall 2011) and post-intervention (Spring 2012). The intervention (January-April 2012) consisted of: policy implementation, professional development training, on-site booster sessions and ongoing technical assistance.

MVPA was classified as the “vigorous” SOPLAY category. Random-effects regression models examined the impact of the intervention on the proportion of boys/girls observed in MVPA or sedentary. A total of 4,525 observations were collected. At baseline, 16.1% and 11.4% of boys and girls, respectively, were engaged in MVPA compared to 20.1% and 17.2% at post-intervention. Additionally, at post-intervention, observations demonstrated a decrease of ~10% in sedentary behavior for boys and girls The comprehensive and coordinated approach implemented over 4 months can lead to important changes in the proportion of children in MVPA while also reducing sedentary behavior. Further exposure to the comprehensive approach has the potential to help ASPs meet MVPA goals outlined in polices.

Barron's Medical Journal Interviewed one of Houston Leading Authorities in the Geneis 129 Diet, Jan Jordan a Nutritionist, Jan has taught diet & lifestyle from a biblical prospective for over 12 years & is a Naturopathic Doctor, Certified Nutritionist, Certified Christian Counselor, Raw Food Chef and Halleluhjah Acres Health Minister. She can be contacted at janjordannd@yahoo.com

Jan says we must start with the quote: And God said, Behold, I have given you every herb bearing seed, which is upon the face of all the earth, and every tree, in the which is the fruit of a tree yielding seed; to you it shall be for meat.

Selenium is at the core of this nontoxic cocktail that gently triggers cancer cells to initiate programmed cell death. The specific form of selenium, sodium selenite has been shown in many scientific studies to be able to kill cancer cells while causing no harm to normal healthy cells. This is an enormously important discovery because conventional chemotherapy kills many more normal healthy cells than cancer cells. Two other components of the nutraceutical cocktail act synergistically allowing very low doses of sodium selenite and a very simple schedule of administration.

Two minerals, zinc and selenium, are key in maintaining balance in the body and keeping cancer away. Recent research has added to the pile of data underscoring the importance of these minerals in keeping women cancer-free.

The identification and characterization of functional elements in eukaryotic genomes, primarily using selenium-dependent genes can help breast cancer patients. On the one hand, we develop and apply computational tools to identify selenoproteins and other genomic features. On the other, we use population genetics and molecular evolution approaches to study their evolution. Because nutrition is a prominent selective force in humans and other primates, we are particularly interested in whether differences in dietary selenium have shaped the use and regulation of selenoproteins in these species. We are also interested in more theoretical aspects of sequence evolution

Jan goes on to explain around the year 1999 she went to her primary care Physician and took a Mammogram and was advised to see a Oncologist. Jan decided to take matters in here hands and treat the lump on her breast without consulting a Oncologist. Jan a nutritionist by education started on the Genesis 129 Diet which is just food with seeds and six months later she says that she was cancer free. It is now 14 years later and still cancer free says that this diet works for her and many other folks. The area of concern is that people that start the diet with stage1 or stage 2-breast cancer and go back to eating their normal diet and this is when the breast cancer reoccurs.

Barron’s Medical Journal decided to look in to the science behind the Genesis 129 diet. The association between dietary fat and breast cancer has been examined for a long time; it remains one of the most controversial in nutritional epidemiology (In the early 1940s, animal studies suggested that high-fat diets could stimulate mammary carcinogenesis. This notion was subsequently reinforced by results of international correlation studies and migrant studies; the latter showed that migrant populations that replaced their indigenous low-fat diets with high-fat, Western diets experienced breast cancer incidence rates similar to those of the host populations. Beginning in the mid-1970s, case–control study reports tended to show positive associations between dietary fat intake and breast cancer.

However, the possibility of differential recall of diet between case and control subjects cast doubt on these results Although pooled analyses of subsequent cohort studies failed to show an association between fat intake and the risk of breast cancer a more recent meta-analysis of 14 cohort studies that adjusted for energy intake found that women who consumed the highest levels of total fat had a statistically significant 13% higher risk of breast cancer than those who consumed the lowest levels. Recently reported findings from the Women’s Health Initiative (WHI) Randomized Controlled Dietary Modification Trial provided suggestive evidence that a low-fat diet can reduce the incidence of breast cancer incidence, although follow-up was shorter and the reduction in fat intake in the intervention group, smaller than originally planned. The Geneis diet provided the women with 650 calories (kcal) in the form of two pints of semi-skimmed milk, four portions of vegetables (80g/portion), one portion of fruit, a salty low calorie drink and a multivitamin and mineral supplement. God instructs man to eat (Genesis 1:29). The dense living nutrients found in raw foods and their juices produce abundant energy and vibrant health while satisfying our cells’ nutritional needs, controlling hunger efficiently.

What a great conference so many of the major food companies are spending billions on research that focus on how we eat and what we eat can help with breast cancer.

Tuesday, October 8, 2013

How Susan G. Komen And Personalized Medicine--Genomics Is A Breast Cancer Game Changer


Barron’s Medical Journal Reporting From Boston At Harvard University Beth Israel Medical Center

How Susan G. Komen And Personalized Medicine--Genomics Is A Breast Cancer Game Changer


Boston MA ( AP )Barron’s Medical Journal investigative report on breast cancer fundraising series starts with The Susan G Komen March. With only fifteen percent of the research dollars spent on

Brought To You By Singer & Song Writer Kristen Mills Personalized Medicine, Barron's wants to make all of the new patient’s that is going to get to go the Doctors now that they have Affordable Health Care Act ( Obama Care ) BMJ want to make sure that patients are asking about genomics for breast cancer. DNA methylation patterns can discover three hundred times faster than a mammogram can breast cancer. Breast cancer is one of the most prevalent human malignancies and is a major cause of cancer-related morbidity and mortality. Invasive ductal carcinoma (IDC) of the breast is a phenotypically diverse disease, consisting of tumors with varying pathologic and molecular characteristics. The primary biological subtypes of IDC include estrogen receptor (ER)– and progesterone receptor (PR)–positive tumors (luminal A and B), tumors that are human epidermal growth factor receptor 2 (HER2)–enriched, and tumors that are ER/PR-negative (basal-like). These molecular determinants have significant effects on metastatic behavior and clinical outcome. For example, ER/PR+tumors are generally associated with better clinical prognosis, whereas basal-like (ER/PR− and HER2−, triple-negative) tumors are associated with higher rates of metastasis and death The genomic alterations, including both genetic and epigenetic aberrations, underlying these differing metastatic potentials are ill-defined.

Significant effort has been undertaken to more accurately define the molecular alterations underlying breast cancer. For example, it has been shown that hormone receptor (HR) status is prognostic for clinical outcome. Mutations in genes such asBRCA1, PTEN, and PIK3CA help promote breast cancer oncogenesis and are enriched in specific subgroups of IDC Genome-wide sequencing surveys have been performed to identify the scope of mutations in breast cancers). These data demonstrate that there exists substantial biological heterogeneity between and within the ER/PR+ and ER/PR− subgroups for which the molecular foundations remain obscure In addition, gene expression classifiers have been developed to help predict metastatic risk). Despite their increasing use in the clinic, the genomic root causes of the transcriptome differences that underlie metastatic potential are unclear.

It is well established that widespread changes in DNA methylation patterns occur during oncogenesis and tumor progression Cancer-specific changes in DNA methylation can alter genetic stability, genomic structure, and gene expression). Promoter CpG island methylation can result in transcriptional silencing and plays an important role in the oncogenic process A CpG island methylator phenotype (CIMP), which is associated with a strong tendency to hypermethylate specific loci, has been described in a subset of colorectal cancers, and recently in a subgroup of gliomas Aberrations in DNA methylation have been reported in human breast cancer, but the impact of the methylome on metastasis and the presence of a B-CIMP have remained elusive To resolve these questions, we conducted a systematic, genome-wide characterization of the breast cancer methylome in breast cancers with diverse metastatic behavior.

Cancer cells undergo massive alterations to their DNA methylation patterns that result in aberrant gene expression and malignant phenotypes. However, the mechanisms that underlie methylome changes are not well understood nor is the genomic distribution of DNA methylation changes well characterized.

Here, we performed methylated DNA immunoprecipitation combined with high-throughput sequencing (MeDIP-seq) to obtain whole-genome DNA methylation profiles for eight human breast cancer cell (BCC) lines and for normal human mammary epithelial cells (HMEC). The MeDIP-seq analysis generated non-biased DNA methylation maps by covering almost the entire genome with sufficient depth and resolution. The most prominent feature of the BCC lines compared to HMEC was a massively reduced methylation level particularly in CpG-poor regions. While hypomethylation did not appear to be associated with particular genomic features, hypermethylation preferentially occurred at CpG-rich gene-related regions independently of the distance from transcription start sites. We also investigated methylome alterations during epithelial-to-mesenchymal transition (EMT) in MCF7 cells. EMT induction was associated with specific alterations to the methylation patterns of gene-related CpG-rich regions, although overall methylation levels were not significantly altered. Moreover, approximately 40% of the epithelial cell-specific methylation patterns in gene-related regions were altered to those typical of mesenchymal cells, suggesting a cell-type specific regulation of DNA methylation. Cancer-specific alterations in DNA methylation are hallmarks of human malignancies; however, the nature of the breast cancer epigenome and its effects on metastatic behavior remain obscure. To address this issue, we used genome-wide analysis to characterize the methylomes of breast cancers with diverse metastatic behavior. Groups of breast tumors were characterized by the presence or absence of coordinate hypermethylation at a large number of genes, demonstrating a breast CpG island methylator phenotype (B-CIMP).

The B-CIMP provided a distinct epigenomic profile and was a strong determinant of metastatic potential. Specifically, the presence of the B-CIMP in tumors was associated with low metastatic risk and survival, and the absence of the B-CIMP was associated with high metastatic risk and death. B-CIMP loci were highly enriched for genes that make up the metastasis transcriptome. Methylation at B-CIMP genes accounted for much of the transcriptomal diversity between breast cancers of varying prognosis, indicating a fundamental epigenomic contribution to metastasis. Comparison of the loci affected by the B-CIMP with those affected by the hypermethylator phenotype in glioma and colon cancer revealed that the CIMP signature was shared by multiple human malignancies. Our data provide a unifying epigenomic framework linking breast cancers with varying outcome and transcriptomic changes underlying metastasis.

Semiconductor Sequencing Chips that create a direct connection between Biochemical and digital information, bringing these two languages together.Hybrid's chips are designed like any other semiconductor chips.

Pairing proprietary semiconductor technology with sequencing chemistry a nucleotide is incorporated into a strand of DNA by a polymerase, a hydrogen ion is released. Hybrid Pharma used a high-density array of micro-machined wells for bioctechnology process in a massive way. Each well holds a different DNA template. Beneath the wells is an ion-sensitive layer and beneath that a proprietary Ion sensor.

When a nucleotide, is added to a DNA template and is then incorporated into a strand of DNA, a hydrogen ion will be released. The charge from that ion will change the pH of the solution, which can be detected. Hybrid's sequencer—essentially will call the base, going directly from Biochemical information to digital information.

If there are two identical bases on the DNA strand, the voltage will be double, and the chip will record two identical bases called.

This process uses no scanners, no cameras, no light—each Nucleotide incorporation is recorded in a real time process.

Panoincell uppressed p53, as it is in many cancers, defective cells multiply, fueling Breast Cancer. p53 can't order a bad cell to kill itself without p63 and p73 also being active.

When metastatic Breast Cancer occurs p63 is inactive.The reactivation of TAp63 could benefit patients with metastatic breast cancer. Make sure you ask your Marcher how much of the money raised at the Susan G. Komen March go to Genomic Science.

Tuesday, October 1, 2013

Breast Cancer Is The Number One Reason Affordable Care Act (ACA) Must Stay In Place Congress


Barron’s Medical Journal Reporting From The Texas State Fair In Dallas Texas USA At The Cotton Bowl The Sight For This Year’s Prairie View A&M University Vs. Grambling State University NCAA Football Game.

Breast Cancer Is The Number One Reason Affordable Care Act (ACA) Must Stay In Place Congress:


Dallas Texas ( AP ) Barron’s Medical Journal is in Dallas asking the question is the team and fans of the Prairie View A&M University Vs Grambling State University Game How many people attending the game will have a relatives that will get breast cancer. Research says it is eighteen percent. Affordable Care Act (ACA) by design is to reduce the rate of eighteen percent in the African American Communities to fewer than five percent of patients that will die one year after diagnoses.

Affordable Care Act (ACA) or Obamacare, is a United States federal statute signed into law by President Barack Obama on March 23, 2010. Together with the Health Care and Education Reconciliation Act

On October 1, 2013 The Health Insurance Marketplace is open! You can now enroll in a health insurance plan that covers essential benefits, pre-existing conditions, and preventive services. Most people who apply for coverage will qualify for lower costs of some kind in the Marketplace. Four things you can do before you apply.

1. Find out how the Marketplace works.

2. Learn how to choose the health insurance plan that’s right for you.

3. Get personal help applying for health coverage.

4. Ask us a question about the Marketplace by phone or live web chat.

Choose a plan that meets your needs and enroll, Coverage starts as soon as January 1, 2014.

Barron’s Medical Journal Ask Rose Conrad Ph.D. C.E.O Of Sam Houston Biotech to talk about how breast cancer works to destroy a patients life. Conrad says that no one dies of breast cancer only when breast cancer spreads to your lungs. If you get a breast cancer genomics test by the age of forty you have a ninety four percent chance to live to sixty. Conrad gose on to say Breast Cancer and HIF-1 Is Why ObamaCare must stay in place. To stop breast cancer from spreading to the patients lungs. Breast cancer spreading to the lungs is the number one reason breast cancer patients expire. Primary Care Physicians and Obama Care is key to prevent women expiring from breast cancer and extend their life.

Sam Houston Biotechnology Scientist Using Genomics has made progress in Houston working with HIF-1. Hypoxia-inducible factor 1 (HIF-1) is a transcriptional activator forbreast cancer. Advances in biochemical purifica- tion, molecular characterization and cellular - molecular biol- ogy ofHIF-1. Sam Houston found involvement in human breast cancer progression, based on the analysis of human breast cancer biopsies and experimental animal mice models. HIF-1 as a therapeutic target can extend the life of many stage four breast Cancer patients.

Sam Houston discovered The tumor suppressors VHL (von Hippel-Lindau protein) and p53 target HIF-1α for ubiquitination to inactivation breast cancer tumors cells increases the of HIF-1. This process Increased phosphatidylinositol 3-kinase (PI3K) and AKT. In breast cancer, increased activity of the HER2 (also known as neu) receptor tyrosine kinase is associated with increased tumor grade, chemotherapy resistance, and decreased patient survival. HER2 has also been implicated as an inducer of VEGF expression. Sam Houston demonstrate that HER2 signaling induced by overexpression in mouse 3T3 cells or human MCF-7 breast cancer cells results in increased HIF-1α protein and VEGF mRNA expression that is dependent upon activity of PI3K, AKT (also known as protein kinase B), and the downstream kinase FRAP (FKBP-rapamycin-associated protein)

Sam Houston concern with patients with diabetes and breast cancer may not receive full Benefit of HIF-1. All women having access to a physician can have access to HIF-1. Genomics provide a faster cheaper more effective way to detecting Breast Cancer by using Semiconductor Sequencing. A example of this technique is Sam Houston Semiconductor Sequencing. "Quantum Theory" In Action for Breast Cancer Patients.

A polymerase is an enzyme whose central function is associated with polymers of nucleic acids such as RNA and DNA. The primary function of a polymerase is the polymerization of new DNA or RNA against an existing DNA or RNA template in the processes of replication and transcription. In association with a Sam Houston also uses a Visualize Real-Time Breast Cancer Data using Signal Stochastic Resonance Units Neurons Detection and Analysis for Breast Cancer model after McCulloch-Pitts. Gennxeix. computer-assisted diagnosing of breast cancer from mammograms. Sam Houston works is a genetic network simulation trained with tumor incidence data from knockout experiments.

Sam Houston uses Semiconductor Sequencing Chips that create a direct connection between Biochemical and digital information, bringing these two languages together. Gennxeix's chips are designed like any other semiconductor chips.

Pairing proprietary semiconductor technology with sequencing chemistry a nucleotide is incorporated into a strand of DNA by a polymerase, a hydrogen ion is released. Sam Houston used a high-density array of micro-machined wells for bioctechnology process in a massive way. Each well holds a different DNA template. Beneath the wells is an ion-sensitive layer and beneath that a proprietary Ion sensor.

Genomics can be the GPS to Extend life in Breast Cancer Patients.